Duodenal+Ulcer

This condition results from the digestive action of acid gastric juice on the mucosal lining of the distal esophagus, stomach (usually lesser curvature), and upper portion of the duodenum. Duodenitis is a non-specific inflammation of the duodenum and represents an early phase of the acid attack on the gastric mucosa. It frequently progresses to peptic ulcer formation. The basic problem and pathophysiologic process is, therefore, peptic ulcer formation.

Orgonomic Perspective of Etiology
The reason for the failure of the mucosa to withstand acid attack in certain individuals prone to develop peptic ulcer (the predisposition to illness) is, in the vast majority of cases, not understood by classical medicine. Contrary to popular belief, there is no hard evidence that dietary factors cause, perpetuate, or aggravate this condition (5). Reich showed that chronic sympatheticotonia, with its tendency toward increased acid production in the stomach and spasm of the intestinal wall, is only the somatic aspect of peptic ulcer disease, while repressed hatred is its psychic counterpart. The basis for both the psychic and somatic component of this illness is a chronic energetic contraction of the diaphragmatic segment. This is the biophysical basis for what is commonly referred to as the predisposition to ulcer formation. With armoring of the diaphragmatic segment, the mucosal lining of the organs contained in this segment becomes contracted with a restriction of blood. Vasospasm leads to greater mucosal susceptibility to ulcer formation with the final sequellae of perforation and hemorrhage. Sympatheticotonia consists of a severe energetic contraction of the diaphragmatic segment. Chronic sympathetic excitation in this illness contains enormous quantities of repressed hatred. Only by expressing hatred from this segment can the "predisposition" for ulcer formation be eliminated.[|1]